Influenzanet is a system to monitor the activity of influenza-like-illness (ILI) with the aid of volunteers via the internet

http://www.influenzanet.eu/

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Developing the framework for an epidemic forecast infrastructure.
http://www.epiwork.eu/

The Seventh Framework Programme (FP7) bundles all research-related EU initiatives.

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Participating countries and volunteers:

The Netherlands 12261
Belgium 3995
Portugal 2149
Italy 0
Great Britain 3342
Sweden 8763
Austria 559
Switzerland 162
France 0
Spain 1012
Ireland 351
Denmark 2917
InfluenzaNet is a system to monitor the activity of influenza-like-illness (ILI) with the aid of volunteers via the internet. It has been operational in The Netherlands and Belgium (2003-2017), Portugal (since 2005) and Italy (since 2008), and the current objective is to implement InfluenzaNet in more European countries.

In contrast with the traditional system of sentinel networks of mainly primary care physicians coordinated by the European Influenza Surveillance Scheme (EISS), InfluenzaNet obtains its data directly from the population. This creates a fast and flexible monitoring system whose uniformity allows for direct comparison of ILI rates between countries.

Any resident of a country where InfluenzaNet is implemented can participate by completing an online application form, which contains various medical, geographic and behavioural questions. Participants are reminded weekly to report any symptoms they have experienced since their last visit. The incidence of ILI is determined on the basis of a uniform case definition.

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H5N1 Influenza virus targets human pulmonary endothelial cells

H5N1 Influenza virus targets human pulmonary endothelial cells

The case fatality rate for H5N1 influenza virus infections in humans is considerably higher than that for seasonal influenza virus infections. Respiratory failure due to acute respiratory distress syndrome is a complication of severe H5N1 disease.

Zeng et al. show that, unlike seasonal influenza viruses, H5N1 influenza virus can target human pulmonary endothelial cells for efficient replication, resulting in high virus loads and an overwhelming immune response. These results shed light on the pathogenic mechanisms of pulmonary endothelial injury associated with H5N1 influenza virus infections. CONTINUE READING

Nov. 9, 2011, midnight